Measurements

ID experiment control background genotype manipulation diet temperature start mean median max num
120 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol D1014.3 RNA Interference
119 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol F26H9.6 RNA Interference
118 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol C52E4.4 RNA Interference
116 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol Y40B1A.4 RNA Interference
115 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol C01H6.5 RNA Interference
113 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol F28D9.1 RNA Interference
112 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol F26F4.11 RNA Interference
111 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol T09B4.10 RNA Interference
110 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol F29G9.3 RNA Interference
109 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol F10B5.6 RNA Interference
108 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol R02D3.3 RNA Interference
107 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol N2 Bristol RNA Interference
114 Table 2. First-order interactors of LAGs in the HLN regulate lifespan in C. elegans. N2 Bristol F18A1.5 RNA Interference
183 Figure 5: Hac1-independent resistance to tunicamycin in afg3∆ and rpl20bΔ cells wild-type 30.0 23.2 24.0 38.0 40
184 Figure 5: Hac1-independent resistance to tunicamycin in afg3∆ and rpl20bΔ cells afg3 30.0 28.4 28.0 56.0 160
185 Figure 5: Hac1-independent resistance to tunicamycin in afg3∆ and rpl20bΔ cells hac1 30.0 25.6 26.0 40.0 20
186 Figure 5: Hac1-independent resistance to tunicamycin in afg3∆ and rpl20bΔ cells afg3;hac1 30.0 30.7 32.0 50.0 40
136 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism sir2;fob1;afg3 30.0 29.1 30.0 45.0 40
137 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism sir2;fob1 30.0 30.8 34.0 46.0 20
138 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism sir2;fob1;afg3 30.0 46.0 44.0 78.0 20
139 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism sir2;fob1 Dietary Restriction 30.0 42.9 50.0 64.0 20
140 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism sir2;fob1;afg3 Dietary Restriction 30.0 45.3 48.0 67.0 20
133 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism wild-type 30.0 20.3 19.0 38.0 80
132 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism afg3;fob1 30.0 38.3 40.0 60.0 40
131 Figure 6: Deletion of AFG3 reduces cytoplasmic translation and extends life span by a Sir2-independent, Gcn4-dependent mechanism fob1 30.0 32.7 35.0 52.0 39